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Phosphorylation of AMPK increased after the suppression of O-GlcNAcylation. We found that O-GlcNAcylation of AMPK suppressed the activity of this regulator, thereby inhibiting ULK1 activity and autophagy. Conclusion We characterized a new function of O-GlcNAcylation in the suppression of autophagy via regulation of AMPK. Graphical abstract Blockage of O-linked GlcNAcylation induces AMPK dependent autophagy in bladder cancer cells. © The Author(s) 2020.Objective This study reports a Chinese patient with a Congenital Disorder of Glycosylat
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