https://www.selleckchem.com/pr....oducts/tipranavir.ht
, and amplification/mutation showed tendency toward mutual exclusivity. Copy-number alteration load was associated with likelihood to recur. Mutational signatures were dominated by aging and APOBEC activation and lacked evidence of ultraviolet radiation. HER2 IHC/fluorescence analysis validated amplification but was underpowered to detect mutations. Tumor heterogeneity in terms of amplification status was observed in some cases. Our comprehensive, unbiased analysis shows EMPD is characterized by alterations involving the PI3K-AKT pat
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