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324, both p less then 0.001; HCM r = 0.157, p = 0.05). Native T1 and T2 were similarly correlated in all CKD stages (S3 r = 0.501, S4 0.586, S5 r = 0.424, p less then 0.001 for all). Native T1 was the strongest myocardial discriminator between patients and controls (area under the curve, AUC HCM 0.97; CKD 0.97, HTN 0.98), native T2 between CKD vs HCM (AUC 0.9 and native T1 and T2 between CKD vs HTN (AUC 0.83 and 0.80 respectively), p less then 0.001 for all. CONCLUSIONS Our findings reveal different CMR signatures of common hyp