https://www.selleckchem.com/pr....oducts/durvalumab.ht
ical interaction with and synaptic trafficking of NMDARs in the hypothalamus. Inhibiting α2δ-1, blocking NMDARs, or disrupting α2δ-1-bound NMDARs reversed chronic stress-induced sympathetic outflow and persistent hypertension. Thus, α2δ-1-dependent NMDAR activity in the hypothalamus is an effector of genetic-environment interactions and may be targeted for treating stress-induced neurogenic hypertension.Distributed population codes are ubiquitous in the brain and pose a challenge to downstream neurons that must learn an appropriate r
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