https://www.selleckchem.com/products/wz-811.html
These results suggest that the suppression of Rad53 activation is a key event in initiating a meiotic program that repairs programmed DSBs.The crosstalk between hematopoietic stem/progenitor cells (HSC), both normal and leukemic, and their neighboring bone marrow (BM) microenvironment (niche) creates a reciprocal dependency, a master regulator of biological process, and chemotherapy resistance. In acute myeloid leukemia (AML), leukemic stem/progenitor cells (LSC) anchored in the protective BM microenvironment, reprogram and transform thi
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